Recombinant human WNT7A   

Protein Wnt-7a; wnt 7a;Protein Wnt-7a precursor; proto-oncogene wnt7a protein; wingless-type MMTV integration site family, member 7A; WNT7A; WNT7A_HUMAN.  

濃度：1mg/ ml

來源：Recombinant Human

純度：≥95% SDS-PAGE

表達系統：Escherichia coli

標簽：His tag   

蛋白長度：Full length protein

內毒素水平：<1.000 Eu/µg

純化方法：HPLC

應用：SDS-PAGE，Western blot，ELISA

Biological activity,immunology research

保存：-20℃

保質期：1年

Ligand for members of the frizzled family of seven transmembrane receptors. Probable developmental protein. Signaling by Wnt-7a allows sexually dimorphic development of the mullerian ducts.Defects in WNT7A are the cause of limb pelvis hypoplasia aplasia syndrome (LPHAS) [MIM:276820]. A syndrome of severe deficiency of the extremities due to hypo- or aplasia of one or more long bones of one or more limbs. Pelvic manifestations include hip dislocation, hypoplastic iliac bone and aplastic pubic bones. Thoracic deformity, unusual facies and genitourinary anomalies can be present.

產品名稱：Rabbit Anti-WNT7A antibody

Rabbit Anti-WNT7A 

別名：Protein Wnt-7a; wnt 7a;Protein Wnt-7a precursor; proto-oncogene wnt7a protein; wingless-type MMTV integration site family, member 7A; WNT7A; WNT7A_HUMAN.            

來源：Rabbit

克隆類型：Polyclonal

濃度：1mg/ml

亞型：IgG

反應：Human,Mouse,Rat

應用： WB=1:1000-1:2000,Elisa=1:1000-1:2000,IHC-P=1:100-500,IHC-F=1:100-500,ICC/IF=1:100-500,IF=1:100-500

理論分子量：41kDa

免疫原：KLH conjugated synthetic peptide derived from human WNT7A

保存：-20℃
保質期：1年

產品名稱：Anti-WNT7A antibody

Mouse Anti-WNT7A

別名Protein Wnt-7a; wnt 7a;Protein Wnt-7a precursor; proto-oncogene wnt7a protein; wingless-type MMTV integration site family, member 7A; WNT7A; WNT7A_HUMAN.               

來源：Mouse

克隆類型：Monoclonal

濃度：1mg/ml

亞型：IgG

反應：Human

應用： WB=1:1000-1:2000,Elisa=1:1000-1:2000,IHC-P=1:100-500,IHC-F=1:100-500,ICC/IF=1:100-500,IF=1:100-500  

反應:  Human

理論分子量：41kDa

免疫原：KLH conjugated synthetic peptide derived from human WNT7A

保存：-20℃
保質期：1年

Ligand for members of the frizzled family of seven transmembrane receptors. Probable developmental protein. Signaling by Wnt-7a allows sexually dimorphic development of the mullerian ducts.Defects in WNT7A are the cause of limb pelvis hypoplasia aplasia syndrome (LPHAS) [MIM:276820]. A syndrome of severe deficiency of the extremities due to hypo- or aplasia of one or more long bones of one or more limbs. Pelvic manifestations include hip dislocation, hypoplastic iliac bone and aplastic pubic bones. Thoracic deformity, unusual facies and genitourinary anomalies can be present.